Atherosclerosis lesion progression during inhalation exposure to environmental tobacco smoke: a comparison to concentrated ambient air fine particles exposure

Chen, LC; Quan, C; Hwang, JS; Jin, X; Li, Q; Zhong, M; Rajagopalan, S; Sun, Q

HERO ID

1077134

Reference Type

Journal Article

Year

2010

Language

English

PMID

20235771

HERO ID 1077134
In Press No
Year 2010
Title Atherosclerosis lesion progression during inhalation exposure to environmental tobacco smoke: a comparison to concentrated ambient air fine particles exposure
Authors Chen, LC; Quan, C; Hwang, JS; Jin, X; Li, Q; Zhong, M; Rajagopalan, S; Sun, Q
Journal Inhalation Toxicology
Volume 22
Issue 6
Page Numbers 449-459
Abstract Environmental tobacco smoke (ETS) and ambient air fine particulate matter (PM(2.5)) are both complex mixtures that have important adverse effects on the cardiovascular system. Although exposures to these complex mixtures have been studied individually, direct comparisons between the two has not been performed. In this study, the authors employed a novel, noninvasive ultrasound biomicroscopy method (UBM) to assess the effects of long-term, low-concentration inhalations of side-stream smoke (SS) and concentrated ambient PM(2.5) (CAPs) on plaque progression. ApoE(-/-) mice (n = 8/group) on high-fat chow (HFC), or normal chow (NC), were exposed to SS (PM = 450 microg/m(3)) or filtered air (FA) for 6 h/day, 5 days/week, for 6 months; CAPs exposure was at 134 microg/m(3) (NC only). Mortality during the SS exposure was greater in the HFC than in the NC, and SS significantly enhanced the effects of diet. No mortality was observed in CAPs-exposed mice. At 4 and 6 months, SS produced the greatest change in plaque area in the left common carotid artery (CCA) in HFC as compared to FA or NC, but not in the brachiocephalic artery. In contrast, CAPs exposure significantly enhanced plaque areas in brachiocephalic and left CCA at 3 and 6 months of exposure. The effect of SS was comparable in magnitude to that produced by CAPs at an average PM(2.5) mass concentration that was only 30% as high. In light of the employment of the same animal model, uniform inhalation exposure protocols, time schedules, a noninvasive monitoring protocol, and a parallel study design, these findings have broad applicability.
Doi 10.3109/08958370903373845
Pmid 20235771
Wosid WOS:000280535400002
Is Certified Translation No
Dupe Override No
Comments Source: Web of Science 000280535400002
Is Public Yes
Language Text English
Keyword Concentrated ambient particles; environmental tobacco smoke; cardiovascular disease; atherosclerosis ultrasound imaging
Is Qa No