Rat liver microsomal metabolism of propyl halides

Tachizawa, H; MacDonald, TL; Neal, RA

HERO ID

1737968

Reference Type

Journal Article

Year

1982

Language

English

PMID

7155131

HERO ID 1737968
In Press No
Year 1982
Title Rat liver microsomal metabolism of propyl halides
Authors Tachizawa, H; MacDonald, TL; Neal, RA
Journal Molecular Pharmacology
Volume 22
Issue 3
Page Numbers 745-751
Abstract The in vitro metabolism of 1-propyl halides (chloride, bromide, and iodide) by hepatic microsomes from phenobarbital-induced rats was examined. The following metabolites were detected: propene, 1,2-epoxypropane, 1,2-propanediol, propionic acid, and undefined species bound to protein (for propyl chloride). The addition of exogenous glutathione to the incubation mixture led to the production of S-(1'-propyl)glutathione and S-(2'-hydroxy-1'-propyl)glutathione. The ratio of the metabolites resulting from C1-C2 functionalization [propene, 1,2-propanediol, and S-(2'-hydroxy-1'-propyl)glutathione] to that resulting from C1 functionalization (propionic acid) increased as the halide progressed down the halide order chloride bromide, and iodide. Mechanisms which rationalize the distribution of propyl halide metabolites as a function of the halide are discussed. The preferred mechanism interprets that the results obtained are a consequence of the partitioning of the initial metabolic transformation between alpha-hydroxylation and halogen oxygenation pathways.
Pmid 7155131
Wosid WOS:A1982PQ84000030
Url http://molpharm.aspetjournals.org/content/22/3/745.abstract
Is Certified Translation No
Dupe Override No
Comments Authoring Organization: National Board of Labour Protection (Finland) HEEP COPYRIGHT: BIOL ABS. The in vitro metabolism of 1-propyl halides (chloride, bromide and iodide) by hepatic microsomes from phenobarbital-induced rats was examined. The following metabolites were detected: propene, 1,2-epoxypropane, 1,2-propanediol, propionic acid and undefined species bound to protein (for propyl chloride). The addition of exogenous glutathione to the incubation mixture led to the production of S-(1'-propyl)glutathione and S-(2'-hydroxy-1'-propyl)glutathione. The ratio of the metabolites resulting from C1-C2 functionalization (propene, 1,2-propanediol and S-(2'-hydroxy-1'-propyl)glutathione) to that resulting from C1 functionalization (propionic acid) increased as the halide progressed down the halide order: chloride bromide and iodide. Mechanisms which rationalized the distribution of propyl halide metabolites as a function of the halide were discussed. The preferred mechanism interpreted the results as a consequence of the partitioning of the initial metabolic transformation between alpha-hydroxylation and halogen oxygenation pathways. (Organohalides are major environmental pollutants.)
Is Public Yes
Language Text English
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