Melatonin administration prevents the nephrotoxicity induced by gentamicin

Ozbek, E; Turkoz, Y; Sahna, E; Ozugurlu, F; Mizrak, B; Ozbek, M

HERO ID

1777965

Reference Type

Journal Article

Year

2000

Language

English

PMID

10759677

HERO ID 1777965
In Press No
Year 2000
Title Melatonin administration prevents the nephrotoxicity induced by gentamicin
Authors Ozbek, E; Turkoz, Y; Sahna, E; Ozugurlu, F; Mizrak, B; Ozbek, M
Journal BJU International
Volume 85
Issue 6
Page Numbers 742-746
Abstract <strong>OBJECTIVE: </strong>To investigate the effect of melatonin on the antioxidant enzyme activity and renal tubular necrosis induced by gentamicin.<br /><br /><strong>MATERIALS AND METHODS: </strong>Twenty-four adult male Sprague-Dawley rats were divided into three equal groups. In group 1, the rats were injected with vehicle (controls), in group 2 they were injected with gentamicin for 5 days and in group 3 injected with gentamicin plus melatonin for 5 days. At 24 h after the last injection, rats were killed and the renal cortex separated from the medulla. Most of the cortex was homogenized but a small sample was fixed in formaldehyde solution for histological examination by light microscopy. Blood samples were also taken to assess the serum levels of urea, creatinine, Na+, K+ and gamma-glutamyl transpeptidase (gamma-GT); before death, urine samples were analysed for protein content. Crude extracts of the cortex were used to determine lipoperoxides, reduced glutathione (GSH-Px), catalase and superoxide dismutase (SOD). The results were compared using the Mann-Whitney U-test.<br /><br /><strong>RESULTS: </strong>Compared with the controls rats, gentamicin caused hyperproteinuria, an increase in the level of gamma-GT in serum, a marked increase in lipoperoxides and a significant decrease of GSH-Px, catalase and SOD activity in the kidney. In the rats in group 3 there was a marked restoration in lipid peroxidation, GSH-Px, catalase, SOD activity and proteinuria, and in gamma-GT in serum. In rats in group 2 there was widespread tubular necrosis (grade 2-4) but in rats in group 3 there was a marked reduction in the extent of tubular damage. There was no significant difference in serum levels of Na+, K+, blood urea nitrogen and creatinine.<br /><br /><strong>CONCLUSION: </strong>These results indicate that melatonin prevents the tubular necrosis induced by gentamicin in rats, presumably because it is a potent antioxidant and restores antioxidant enzyme activity in the rat kidney.
Doi 10.1046/j.1464-410x.2000.00531.x
Pmid 10759677
Wosid WOS:000086446100030
Url <Go to ISI>://WOS:000086446100030
Is Certified Translation No
Dupe Override No
Comments Source: Web of Science WOS:000086446100030
Is Public Yes
Language Text English
Keyword gentamicin; nephrotoxicity; melatonin; tubular necrosis; antioxidant enzyme