Flurothyl seizure susceptibility in rats following prenatal methylazoxymethanol treatment

Baraban, SC; Schwartzkroin, PA

HERO ID

3828377

Reference Type

Journal Article

Year

1996

Language

English

PMID

8739122

HERO ID 3828377
In Press No
Year 1996
Title Flurothyl seizure susceptibility in rats following prenatal methylazoxymethanol treatment
Authors Baraban, SC; Schwartzkroin, PA
Journal Epilepsy Research
Volume 23
Issue 3
Page Numbers 189-194
Abstract Methylazoxymethanol acetate (MAMac) is a potent teratogenic agent which can produce ectopic cell placement in developing rat brains. In the present study, we evaluated (i) whether prenatal exposure to MAMac results in a lowered seizure threshold to flurothyl and (ii) if there is a correlation between the number of ectopic cells in MAMac-exposed hippocampus and flurothyl-induced seizure latency. In 60 day old (P60) rats exposed to MAMac in utero, the latencies to myoclonic jerk (173 +/- 2.3 s) and forelimb clonus (215 +/- 4.6 s) were significantly shorter than those of controls (200 +/- 6.9 s and 238 +/- 8.8 s, respectively). MAMac also increased the proportion of flurothyl-treated rats that progressed from bilateral forelimb clonus to generalized tonic-clonic seizures (control: 33%; MAMac: 91%). Shorter seizure latencies were associated with an increased number of ectopic pyramidal cells in region CA1/CA2. These results suggest seizure susceptibility is enhanced in an animal model (MAMac) characterized by abnormal neuronal migration.
Doi 10.1016/0920-1211(95)00094-1
Pmid 8739122
Wosid WOS:A1996UL44900002
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English
Keyword ectopic; flurothyl; hippocampus; rat; seizure