Role of myelofibrosis in hematotoxicity of munitions RDX environmental degradation product MNX. 2009 Update

Meyer, SA

HERO ID

630114

Reference Type

Technical Report

Year

2009

HERO ID 630114
Year 2009
Title Role of myelofibrosis in hematotoxicity of munitions RDX environmental degradation product MNX. 2009 Update
Authors Meyer, SA
Publisher Text University of Louisiana at Monroe
City Monroe, LA
Abstract The purpose of this research is to determine mechanisms through which hexahydro-1-nitroso-3,5-dinitro-1,3,5-triazine (MNX), environmental degradation product of munition hexahydro-1,3,5-trinitro-1,3,5-triazine (RDX), causes anemia after acute exposure in the rat and examine whether similar effects were elicited by subchronic exposure. We have hypothesized MNX targets hematopoeitic stem cells and, like other myelosuppre-sive chemicals, will be fibrogenic to bone marrow. During this reporting period, results from subchronic exposure in which rats were treated orally with ¼ LD50 MNX daily for 4 or 6 weeks were: 1) significant increases in granulo-cytes and platelets, but not erythrocytes, at both times, 2) an increase in serum K and decreases in Na, Cl, glucose, and creatinine levels in the absence of effect on body weight gain and serum albumin, 3) increased liver weights and 4) increased megakaryocytes in bone marrow, but not fibrosis as indicated by methenamine silver
Report Number W81XWH-05-1-0537
Url https://ntrl.ntis.gov/NTRL/dashboard/searchResults.xhtml?searchQuery=ADA517261
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Dupe Override No
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