Perinatal butyl benzyl phthalate (BBP) and bis(2-ethylhexyl)phthalate (DEHP) exposures induce antiandrogenic effects in Sprague-Dawley (SD) rats

Parks, LG; Ostby, JS; Lambright, CR; Abbott, BD; Gray, LE, Jr

HERO ID

675373

Reference Type

Journal Article

Year

1999

Language

English

HERO ID 675373
In Press No
Year 1999
Title Perinatal butyl benzyl phthalate (BBP) and bis(2-ethylhexyl)phthalate (DEHP) exposures induce antiandrogenic effects in Sprague-Dawley (SD) rats
Authors Parks, LG; Ostby, JS; Lambright, CR; Abbott, BD; Gray, LE, Jr
Journal Biology of Reproduction
Volume 60
Issue Suppl 1
Page Numbers 153
Abstract The developmental effects of several endocrine disrupting chemicals that act as androgen receptor (AR) antagonists have been described in our laboratory. While Mylchreest et al. (1998) and Gray et al. (1999) reported that BBP and DEHP produced antiandrogenic effects on male sexual differentiation, studies by Lambright et al. (1999) indicated that DBP and DEHP are not AR antagonists. Although some phthalates like BBP are weakly estrogenic in vitro, this mechanism seems unlikely to explain the developmental alterations because the phthalates do not display estrogenicity in vivo. This study was conducted to determine if BBP and DEHP induce reproductive tract malformations and to identify the mechanisms of action. SD rats were dosed by gavage with 750 mg/kg/d of BBP, DEHP or corn oil from gestational day 14 to postnatal day (PND) 3. On PND-2 anogenital distance (AGD), testes weight and in vitro testosterone (T) production were measured. Testes weights and AGD were significantly decreased for both DEHP and BBP exposed pups and the incidence of areolas (PND-13) was increased. Further, T production was reduced by DEHP treatment. These antiandrogenic-like effects may result from reduced androgen production in the fetal Leydig cells and suggests that the testis is the target organ directly affected by perinatal phthalate exposure. It remains to be determined whether these effects are mediated via direct action of the phthalates on the fetal Leydig cells or through alterations of Sertoli cell paracrine secretions.
Wosid WOS:000081141300256
Is Certified Translation No
Dupe Override No
Comments Authoring Organization: ARTHUR D LITTLE INC
Is Public Yes
Language Text English
Keyword Pregnancy; Rats; Animal; Female; Sprague-Dawley; Androgen Antagonists TOXICITY; Phthalic Acids TOXICITY; Diethylhexyl Phthalate TOXICITY; Fetus DRUG EFFECTS; Testis DRUG EFFECTS; Organ Weight DRUG EFFECTS; Testosterone BIOSYNTHESIS; No cas rn; 85-68-7; 117-81-7; 57-85-2
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