Di-iso-nonylphthalate (DINP) metabolites in human urine after a single oral dose of deuterium-labelled DINP

Koch, HM; Angerer, J

HERO ID

680024

Reference Type

Journal Article

Year

2007

Language

English

PMID

17182279

HERO ID 680024
In Press No
Year 2007
Title Di-iso-nonylphthalate (DINP) metabolites in human urine after a single oral dose of deuterium-labelled DINP
Authors Koch, HM; Angerer, J
Journal International Journal of Hygiene and Environmental Health
Volume 210
Issue 1
Page Numbers 9-19
Abstract Di-iso-nonylphthalate (DINP), a complex mixture of predominantly nine-carbon branched chain dialkyl phthalate isomers, has replaced di-(2-ethylhexyl)phthalate (DEHP) as the major plasticiser for polyvinylchloride (PVC) polymers. Similar to DEHP, DINP is a developmental and reproductive toxicant in rodents. This study for the first time describes human metabolism and elimination of DINP in a male volunteer after we applied a single oral DINP dose of 1.27 mg/kg body-weight. To avoid interference by omnipresent background exposure we used deuterium-labelled DINP. We investigated the urinary excretion of the simple monoester mono-iso-nonylphthalate (MINP) and oxidised isomers with hydroxy (OH-MINP), oxo (oxo-MINP) and carboxy (carboxy-MINP) functional groups. We used isomeric MINP and three specific oxidised isomer standards for quantification: mono-(4-methyl-7-hydroxy-octyl)phthalate (7OH-MMeOP), mono-(4-methyl-7-oxo-octyl)phthalate (7oxo-MMeOP) and mono-(4-methyl-7-carboxyheptyl)phthalate (7carboxy-MMeHP). These specific DINP metabolites are currently the only synthetic DINP metabolite standards available. Within 48 h we recovered 43.6% of the applied dose in urine as the above DINP metabolites, 20.2% as OH-MINP, 10.7% as carboxy-MINP, 10.6% as oxo-MINP and only 2.2% as MINP. Other oxidised DINP metabolites not determined in this study probably increase the share of the DINP dose excreted via urine. Elimination followed a multi-phase pattern, elimination half-lives in the second phase (beginning 24h post-dose) can only roughly be estimated to be 12h for the OH- and oxo-MINP-metabolites and 18 h for carboxy-MINP metabolites. After 24h, the carboxy-MINP metabolites replaced the OH-MINP metabolites as the major urinary metabolites. All oxidised DINP metabolites are suitable parameters for biomonitoring human DINP exposure.
Doi 10.1016/j.ijheh.2006.11.008
Pmid 17182279
Wosid WOS:000244592600002
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English
Keyword Human metabolism; Di-iso-nonylphthalate (DINP); Urine; Oxidised metabolites; Biomarkers of exposure
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