Decrease in hepatotoxicity by lead exposure is not explained by its mitogenic response

Calabrese, EJ; Baldwin, LA; Leonard, DA; Zhao, XQ

HERO ID

77651

Reference Type

Journal Article

Year

1995

Language

English

PMID

7782558

HERO ID 77651
In Press No
Year 1995
Title Decrease in hepatotoxicity by lead exposure is not explained by its mitogenic response
Authors Calabrese, EJ; Baldwin, LA; Leonard, DA; Zhao, XQ
Journal Journal of Applied Toxicology
Volume 15
Issue 2
Page Numbers 129-132
Abstract The present research was conducted to evaluate the effect of mitogen pre-exposure on CCl4-induced hepatotoxicity. Male Wistar rats were administered a single i.p. injection of CCl4 (0.3 ml kg-1 in corn oil) 48 h following either a single dose of lead nitrate (0.33 mg kg-1) or distilled water via i.v. injection. Hepatotoxicity, as measured by serum alanine aminotransferase (ALT) and aspartate aminotransferase (AST) levels, was monitored 6, 24, 48, 72 and 120 h after CCl4 exposure. The lead nitrate-pretreated rats displayed markedly lower serum ALT and AST levels at 24, 48 and 72 h than rats pretreated with distilled water. However, treatment with the antimitotic agent colchicine did not alter the lead-induced protection. These findings suggest that the lead-induced protection is not associated with the major mitogenic response of lead, despite its strong temporal association. A critical review of the available toxicological data also argues against the lead protection being a function of its capacity to inhibit cytochrome P-450.
Doi 10.1002/jat.2550150212
Pmid 7782558
Wosid WOS:A1995QP84700010
Is Certified Translation No
Dupe Override No
Comments J. Appl. Toxicol. 15: 129-132.
Is Public Yes
Language Text English
Keyword MITOGENICITY; LEAD NITRATE; HEPATOTOXICITY; CARBON TETRACHLORIDE; WISTAR RAT; COLCHICINE
Is Qa No