Involvement of extracellular oxidative burst in salicylic acid-induced stomatal closure in Arabidopsis

Khokon, AR; Okuma, E; Hossain, MA; Munemasa, S; Uraji, M; Nakamura, Y; Mori, IC; Murata, Y

HERO ID

881261

Reference Type

Journal Article

Year

2011

Language

English

PMID

21062318

HERO ID 881261
In Press No
Year 2011
Title Involvement of extracellular oxidative burst in salicylic acid-induced stomatal closure in Arabidopsis
Authors Khokon, AR; Okuma, E; Hossain, MA; Munemasa, S; Uraji, M; Nakamura, Y; Mori, IC; Murata, Y
Journal Plant, Cell and Environment
Volume 34
Issue 3
Page Numbers 434-443
Abstract Salicylic acid (SA), a ubiquitous phenolic phytohormone, is involved in many plant physiological processes including stomatal movement. We analysed SA-induced stomatal closure, production of reactive oxygen species (ROS) and nitric oxide (NO), cytosolic calcium ion ([Ca²+](cyt)) oscillations and inward-rectifying potassium (K+(in)) channel activity in Arabidopsis. SA-induced stomatal closure was inhibited by pre-treatment with catalase (CAT) and superoxide dismutase (SOD), suggesting the involvement of extracellular ROS. A peroxidase inhibitor, SHAM (salicylhydroxamic acid) completely abolished SA-induced stomatal closure whereas neither an inhibitor of NADPH oxidase (DPI) nor atrbohD atrbohF mutation impairs SA-induced stomatal closures. 3,3'-Diaminobenzidine (DAB) and nitroblue tetrazolium (NBT) stainings demonstrated that SA induced H₂O₂ and O₂⁻ production. Guard cell ROS accumulation was significantly increased by SA, but that ROS was suppressed by exogenous CAT, SOD and SHAM. NO scavenger 2-(4-carboxyphenyl)-4,4,5,5-tetramethylimidazoline-1-oxyl-3-oxide (cPTIO) suppressed the SA-induced stomatal closure but did not suppress guard cell ROS accumulation whereas SHAM suppressed SA-induced NO production. SA failed to induce [Ca²+](cyt) oscillations in guard cells whereas K+(in) channel activity was suppressed by SA. These results indicate that SA induces stomatal closure accompanied with extracellular ROS production mediated by SHAM-sensitive peroxidase, intracellular ROS accumulation and K+(in) channel inactivation.
Doi 10.1111/j.1365-3040.2010.02253.x
Pmid 21062318
Wosid WOS:000287094700006
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English
Keyword [Ca2+](cyt) oscillation; K+(in) channel; NO; peroxidase; ROS
Is Qa No